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In keeping with the reduced capacity of homocysteine-exposed endothelial cells to inhibit platelet aggregation, Lentz and collaborators demonstrated, using experimental hyperhomocysteinemia (induced by a diet depleted of folate, high in methionine, and choline-free) that the endothelial-dependent vasodilation is impaired in hyperhomocysteinemic primates

Interpreting them is no easy task either

This finding contrasts with the results obtained from previous immunophagocytosis experiments

Such breakthrough led to the successful use of bacteria-derived L-asparaginase in treating acute lymphoblastic leukemia (ALL) [76]

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