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doi: 10.1080/14787210.2019.1634545 27 OkomboJ.KiaraS

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While early literature suggested that p53-dependent tumor suppression involved the induction of cell cycle arrest, senescence or apoptosis, the recent analysis of several transactivation-defective p53 mutants has called this model into question and it now appears that alterations in intracellular metabolism may account for the ability of p53 to suppress tumor formation [6870]

It has also been reported that there was a vital change in the Bacteroidetes and Firmicutes at the phyla levels, and Barnesiella , Coprobacter , Lactobacillus , Odoribacter , Intestinimonas , Anaerotruncus , and Staphylococcus at the genus level in the glioma mice during tumour progression [193]