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Taking into account the fact that the stimulation of both receptor subtypes changes the concentration of cAMP, with a decrease in cAMP in the case of the stimulation of A1 receptors and an increase in cAMP in the case of the stimulation of A2A receptors [9], the question arises whether the antioxidant/beneficial action of caffeine could be due to this second messenger

His research has been supported by the Department of Defense and the National Institutes of Health/National Cancer Institute (NCI)

Additionally it also inhibited the levels of (myeloperoxidase) & MDA (malondialdehyde) in the mice epithelium

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