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In laboratory research, IGF-1 LR3 is used to investigate growth factormediated signaling pathways that regulate cellular proliferation, differentiation, metabolism, and protein synthesis

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Chlorogenic acid inhibits the binding of Nrf2 to its repressor protein Keap1 to activate the Nrf2 antioxidant signaling pathway, thereby preventing APAP-induced hepatotoxicity (Wei et al., 2018)

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Using this experimental paradigm, we next determined whether SIRT3 overexpression in HSCs suppresses aging-associated inflammation and tissue function decline by giving rise to progeny that go into the circulation