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Cell-type-specific profiling of human cellular models of fragile X syndrome reveal PI3K-dependent defects in translation and neurogenesis

It appears to accelerate the formation of blood vessels (angiogenesis) and enhance the migration and proliferation of cells involved in tissue regeneration

The severity and duration of deficiency are primary determinantspatients with mild, recently developed deficiency typically respond more rapidly than those with longstanding, severe depletion

(136) adopted a CIA mouse model and an in vitro iTFR cell culture system

The genetic structure of populations from Haiti and Jamaica reflect divergent demographic histories