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recombinant GSTZ1*A protein is relatively resistant to DCA mediated inactivation when compared with the other isoforms dieldrin - a potent reversible inhibitor toward hGSTA1-1, mixed-type inhibition, dieldrin binds specifically to the enzyme presumably at a position that partially overlaps with both the G- and H-site, a site distinct from binding of spiromesifen, binding structure overview ellagic acid - inhibition of isozymes GST A1-1, A2-2, M1-1, M2-2, and P1-1, effects on substrate kinetics, overview Ethacrynic acid ethyl 2-acrylamido-4-phenylthiazole-5-carboxylate - ethyl 2-acrylamido-4-phenylthiophene-3-carboxylate - ethyl 2-acrylamido-5-phenylthiophene-3-carboxylate - ethyl N-acryloyl-N-(4-phenylthiazol-2-yl)glycinate - ferulic acid - inhibition of isozyme GST M1-1 gamma-Glu-Cys(Acm)-Gly 85.2% remaining activity at 0.0003 mM gamma-Glu-Cys(t-buthio)-Gly 85.6% remaining activity at 0.0003 mM gamma-Glu-Met-Gly 79.4% remaining activity at 0.0003 mM gamma-Glu-Thi-Gly 67.4% remaining activity at 0.0003 mM genistein - inhibition of isozymes GST M1-1 and M2-2 glutathione - competitive inhibition by the substrate Gly-Cys-Hyp 87.4% remaining activity at 0.0003 mM Hematin Hyp-Cys-Ala 98.7% remaining activity at 0.0003 mM kaempferol - inhibition of isozymes GST M1-1 and M2-2 L-g-glutamyl-S-[3-([5-[(5-[[5-([5-[(2-carbamimidamidoethyl)carbamoyl]-1-methyl-1H-pyrrol-3-yl]carbamoyl)-1-methyl-1H-pyrrol-3-yl]carbamoyl]-1-methyl-1H-pyrrol-3-yl)carbamoyl]-1-methyl-1H-pyrrol-3-yl]amino)-2-hydroxy-3-oxopropyl]-L-cysteinylglycine - a brostallicin derivative, inhibits GSTP1-1 and GSTM2-2 L-g-glutamyl-S-[3-([5-[(5-[[5-([5-[(2-carbamimidamidoethyl)carbamoyl]-1-methyl-1H-pyrrol-3-yl]carbamoyl)-1-methyl-1H-pyrrol-3-yl]carbamoyl]-1-methyl-1H-pyrrol-3-yl)carbamoyl]-1-methyl-1H-pyrrol-3-yl]amino)-3-oxo-2-(phosphonooxy)propyl]-L-cysteinylglycine - a brostallicin derivative, inhibits GSTP1-1 and GSTM2-2 methyl (E)-4-((4-chloro-3-(N,N-dimethylsulfamoyl)phenyl)-amino)but-2-enoate - methyl N-acryloyl-N-(4-phenylthiazol-2-yl)glycinate - N-((1E)-[4-(dimethylamino)phenyl]methylene)-2-([(1-methyl-1H-pyrrol-2-yl)sulfonyl]methyl)aniline - 78.9% inhibition at 0.1 mM N-((3-methylisoxazol-5-yl )methyl)-N-(4-(4-chloro-2-hydroxylphenyl)thiazol-2-yl)acrylamide - N-((3-methylisoxazol-5-yl)methyl)-N-(4-(2-hydroxylphenyl)-thiazol-2-yl)acrylamide - N-((3-methylisoxazol-5-yl)methyl)-N-(4-(2-methoxyphenyl)-thiazol-2-yl)acrylamide - N-((3-methylisoxazol-5-yl)methyl)-N-(4-(4-fluoro-2-hydroxylphenyl)thiazol-2-yl)acrylamide - N-((3-methylisoxazol-5-yl)methyl)-N-(4-(4-trifluoromethylphenyl)thiazol-2-yl)acrylamide - N-((3-methylisoxazol-5-yl)methyl)-N-(4-phenylthiazol-2-yl)-acrylamide - N-(1-benzyl-1H-pyrazol-4-yl)acrylamide - N-(1-phenyl-1H-pyrazol-4-yl)acrylamide - N-(2-((2-morpholinoethyl)amino)-2-oxoethyl)-N-(4-phenylthiazol-2-yl)acrylamide - N-(2-(cyclohexylamino)-2-oxoethyl)-N-(4-phenylthiazol-2-yl)-acrylamide - N-(2-(oxetan-3-ylamino)-2-oxoethyl)-N-(4-phenylthiazol-2-yl)-acrylamide - N-(2-morpholino-2-oxoethyl)-N-(4-phenylthiazol-2-yl)-acrylamide - N-(4-(2-chlorophenyl)thiazol-2-yl)acrylamide - N-(4-(2-fluorophenyl)thiazol-2-yl)acrylamide - N-(4-(2-hydroxylphenyl)thiazol-2-yl)acrylamide - N-(4-(2-hydroxyphenyl)thiophen-2-yl)-N-((3-methylisoxazol-5-yl)methyl)acrylamide - N-(4-(2-methoxyphenyl)thiazol-2-yl)acrylamide - N-(4-(3,4-dioxylphenyl)thiazol-2-yl)acrylamide - N-(4-(3-chlorophenyl)thiazol-2-yl)acrylamide - N-(4-(4-chlorophenyl)thiazol-2-yl)acrylamide - N-(4-(4-hydroxylphenyl)thiazol-2-yl)acrylamide - N-(4-(4-methoxyphenyl)thiazol-2-yl)acrylamide - N-(4-(4-methylphenyl)thiazol-2-yl)acrylamide - N-(4-(4-trifluoromethylphenyl)thiazol-2-yl)acrylamide - N-(4-(naphthalen-2-yl)thiazol-2-yl)acrylamide - N-(4-bromo-3-(N,N-dimethylsulfamoyl)phenyl)-2-chloroacetamide - - N-(4-chloro-3-(N,N-dimethylsulfamoyl)phenyl)-2-cyanoacetamide - - N-(4-chloro-3-(N,N-dimethylsulfamoyl)phenyl)-2-fluoroacetamide - - N-(4-chloro-3-(N,N-dimethylsulfamoyl)phenyl)-2-oxopropanamide - - N-(4-chloro-3-(N,N-dimethylsulfamoyl)phenyl)acrylamide N-(4-phenyl-5-methyl-thiazol-2-yl)acrylamide - N-(4-phenylthiazol-2-yl)-N-(pyridin-3-ylmethyl)acrylamide - N-(4-phenylthiazol-2-yl)acrylamide - N-(5-phenylthiazol-2-yl)acrylamide - N-acetyl-p-benzoquinoneimine concentration-dependent inhibition N-acryloyl-N-(4-phenylthiazol-2-yl)glycine - N-benzyl-N-(4-phenylthiazol-2-yl)acrylamide - N-ethylmaleimide N-[(1E)-(2-chlorophenyl)methylene]-2-([(1-methyl-1H-pyrrol-2-yl)sulfonyl]methyl)aniline - 88.8% inhibition at 0.1 mM N-[(1E)-(4-chlorophenyl)methylene]-2-([(1-methyl-1H-pyrrol-2-yl)sulfonyl]methyl)aniline - 60.4% inhibition at 0.1 mM N-[(1E)-(4-fluorophenyl)methylene]-2-([(1-methyl-1H-pyrrol-2-yl)sulfonyl]methyl)aniline - 37.1% inhibition at 0.1 mM N-[(1E)-(5-bromo-2-methoxyphenyl)methylene]-2-([(1-methyl-1H-pyrrol-2-yl)sulfonyl]methyl)aniline - 646% inhibition at 0.1 mM N-[3-[(chloroacetyl)(4-nitrophenyl)amino]propyl]-2,2,2-trifluoroacetamide i.e

Keywords: chondrocyte, senescence, autologous chondrocyte implantation, senolytic, FOXO4-DRI Introduction Autologous chondrocyte implantation (ACI) is a biomedical treatment that repairs cartilage injury in the knee joint, which has been shown to reduce pain and facilitate mobility recovery (Kreuz et al., 2019)

Excitatory Amino Similarly, increased release of excitatory amino acids is also involved in nerve function damage after TBI

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