Scientific Communications: intracranial hemorrhage 02.00 - INTRACRANIAL HEMORRHAGE - 02.01 - INTRACEREBRAL HAEMORRHAGE O - ESOC25-1919 THE EFFECT OF EARLY MINIMALLY INVASIVE SURGICAL HAEMATOMA EVACUATION ON PERIHAEMATOMAL OEDEMA FORMATION Maaike Cliteur 1 , Lotte Sondag 2 , Wilmar Jolink 3 , William Peter Vandertop 4 , Jeroen Boogaarts 5 , Marieke Wermer 6 , Ruben Dammers 7 , Floris Schreuder 1 , Karin Klijn 1 1 Department of Neurology, Donders Institute for Brain, Cognition and Behaviour, Radboud University Medical Center, Nijmegen, Netherlands, 2 Department of Neurology, Jeroen Bosch Hospital, 's Hertogenbosch, Netherlands, 3 Department of Neurology, Isala Hospital, Zwolle, Netherlands, 4 Amsterdam UMC, Neurosurgery, Amsterdam Movement Sciences, Vrije Universiteit Amsterdam, Amsterdam, Netherlands, 5 Department of Neurosurgery, Radboud University Medical Centre, Nijmegen, Netherlands, 6 Department of Neurology, University Medical Center Groningen (UMCG), Groningen, Netherlands, 7 Department of Neurosurgery, Erasmus Medical Centre, Erasmus MC Stroke Centre, Rotterdam, Netherlands Background and Aims: In patients with intracerebral haemorrhage (ICH), perihaematomal oedema (PHO) is considered a marker of secondary injury and is independently associated with worse functional outcomes
Pharmacological and clinical applications of cyclophosphamide The FDA indicates that CP is primarily used for the treatment of malignant lymphomas in stages III and IV, as classified by the Ann Arbor staging criteria
39 QuilbeA.MoralsO.BaydounM.KumarA.MustaphaR.MurakamiT.et al (2020)
Detection of nitrotyrosine in aging and osteoarthritic cartilage: Correlation of oxidative damage with the presence of interleukin-1beta and with chondrocyte resistance to insulin-like growth factor 1
In summary, the failure of the endogenous antioxidant defense system allows the massive oxidative insult to trigger a complex array of downstream pathological consequences, including robust inflammatory responses, multiple forms of cardiomyocyte death, and widespread disruption of vital signaling networks
