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The reduced enzymatic activity increases cellular concentrations of both substrates that NNMT would otherwise consume: S-adenosyl-L-methionine (the universal cellular methyl donor) and nicotinamide (the NAD + salvage precursor)

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More recent evidence indicates that sustained GIP receptor agonism in the presence of GLP-1 activation enhances insulin sensitivity and may preferentially reduce visceral adipose tissue

These include some anticonvulsants (medicines used to treat epilepsy), colestyramine, sulfasalazine and methotrexate

Dosage is just one piece of the puzzlebut its the piece that determines whether BPC-157 works for you or not.