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None of these interactions has been quantified in human pharmacokinetic-pharmacodynamic studies

This stabilizes the transcription factor BACH1, which subsequently upregulates angiogenesis-related factors, including PDGFB, EGFR, FGF2, and FGFR1, ultimately driving vascular regeneration

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171 , 172 Additionally, studies on NRC-03 and NRC-07 (two AMPs from the pleurocidin AMP family) showed that the 50% effective concentration (EC 50 ) of cisplatin on tumor cells could be significantly reduced when used in combination with NRC-03 or NRC-07, that is, the cytotoxicity of cisplatin was increased