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To additionally support the contention that the agonists Dihexa and Nle1-AngIV are acting through HGF and its receptor c-Met, hippocampal neurons were transfected with shRNA to knockdown the c-Met receptor

[2] It appears to act at the gene level to increase actin production, whereas TB-500 is an actin-binding protein that appears to sequester actin in areas where its requirement is maximum for cell movement

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Further study is needed to determine the underlying reason for these differences

These figures describe documented research protocols and are offered for research-use-only reference, not as medical advice