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Metformin inhibits chemokine expression through the ampk/nf-b signaling pathway

Preclinical studies in animal models consistently indicate that, within the tens to hundreds of nanometers range, there is a trade-off between circulation time, transvascular extravasation and tissue penetration [38,39,40]

MED12 and HMGA2 are common mutation types in uterine fibroids, with MED12 mutations being the most prevalent [111]

The advantage of these linkers is that they not only maintain the ADCs' drug stability in blood circulation but also enable the rapid release of cytotoxins in the tumor site