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Combining molecular oxygen reduction with energy generation during aerobic metabolism creates the potential of oxidative stress, i.e., damage to critical cellular macromolecules, which may lead to enzyme inactivation, lipid peroxidation, DNA damage, and ultimately, cell death (Regoli and Giuliani, 2014)

Together these investigations have highlighted the therapeutic potential of augmenting the HGF/c-Met system for the treatment oftype 2 diabetes and metabolic syndrome (Fafalios et al., 2011

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