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A growing number of medical conditions are correlated with the increase in the level of reactive oxygen species (ROS), but the pathophysiological mechanisms in which they are involved are beyond the purpose of our paper, and, in addition, their positive effects should not be excluded [4]

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Upstream signaling through PI3K/AKT, MAPK (ERK/JNK/p38), and PKC further enhances Nrf2 stabilization and transcriptional activity

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